|Related Categories||All Alphabetically Sorted, Analytical Standards, Analytical/Chromatography, Chemical Class, Chromatography,|
|grade||certified reference material|
|packaging||pkg of 1 g|
|pharmacopeia traceability||traceable to PhEur P3640000|
|traceable to USP 1576800|
This certified reference material (CRM) is produced and certified in accordance with ISO/IEC 17025 and ISO Guide 34.
Values of analytes vary lot to lot.
Prepare a 0.1M solution in glycerol:PBS (9:1)
Used as an anti-fade reagent in fluorescence microscopy to reduce photobleaching of fluorescent probes such as rhodamine and fluorescein.
An antioxidant that exhibits antimicrobial activity. Propyl gallate has been reported to be an effective antioxidant-based hepatoprotector, both in vitro and in vivo. It has also been shown to prevent neuronal apoptosis and block the death of neurons exposed to FeSO4/GA as well as partially protect endothelial cells against TNF-induced apoptosis. However, propyl gallate induced single strand breaks in DNA at concentrations higher than 0.25 μM when it was combined with copper concentrations at 5 μM and above. Another report, however, found that incubation of isolated rat hepatocytes with propyl gallate at concentrations of >1 mM induced cell killing, whereas, a lower concentration of propyl gallate > 0.5 mM resulted in a ladder formation of soluble low-molecular weight DNA fragments, characteristic of apoptosis.
An antioxidant that exhibits antimicrobial activity. Propyl gallate has been reported to be an effective antioxidant-based hepatoprotector, both in vitro and in vivo. It has also been shown to prevent neuronal apoptosis and block the death of neurons exposed to FeSO4/GA as well as partially protect endothelial cells against TNF-induced apoptosis.
Certificate of Analysis
Astrocytic gap junctional communication decreases neuronal vulnerability to oxidative stress-induced disruption of Ca2+ homeostasis and cell death. Blanc, E.M., et al. J. Neurochem. 70, 958-970, (1998)
Linoleic acid potentiates TNF-mediated oxidative stress, disruption of calcium homeostasis, and apoptosis of cultured vascular endothelial cells. Toborek, M., et al. J. Lipid Res. 38, 2155-2167, (1997)
Aldrich MSDS 1, 1552:C / Arctander, 2710 / Corp MSDS 1 (2), 2967:D / FT-IR 1 (2), 301:A / FT-IR 2 (2), 2854:D / FT-NMR 1 (2), 1261:B / Fenaroli, 698 / IR-Spectra (3), 1032:D / IR-Spectra (2), 903:G / NMR-Reference 2 (2), 295:A / RegBook 1 (2), 1909:K / Sax 6, 2310 / Sigma FT-IR 1 (2), 862:C / Structure Index 1, 306:D:5 / Vapor Phase 3, 1370:B
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