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Puromycin aminonucleoside is used to study human glomerular disease by inducing damage of murine glomerular podocytes1 and is used to study glomerular function and morphology.
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Increasing antibiotic resistance and the lack of new drug development have hindered the success of antibiotics in recent years. Staph aureus is susceptible to a number of antibiotics with various mec...
Selective inhibition of preribosomal RNA synthesis by puromycin aminonucleoside in transformed human fibroblasts: studies of the nature of the inhibition in isolated nuclei and nucleoli. Albanese, E.A. and Studzinski, G.P. J. Cell Physiol. 99, 55-65, (1979)
Systemic administration of naked plasmid encoding HGF attenuates puromycin aminonucleoside-induced damage of murine glomerular podocytes Xuan Bu, Yang Zhou, et al. Am. J. Physiol. Renal Physiol. 301, F784-F792, (2011)
Glomerular function and morphology in puromycin aminonucleoside nephropathy in rats Eva K. M. Löwenborg, et al. Nephrol. Dial. Transplant. 15, 1547-1555, (2011)
A novel nuclear factor κB inhibitor, dehydroxymethylepoxyquinomicin, ameliorates puromycin aminonucleoside-induced nephrosis in mice. Shimo T, Adachi Y, Yamanouchi S, et al. Am. J. Nephrol. 37(4), 302-9, (2013)
Experimentally induced puromycine aminonucleoside nephrosis (PAN) in rats: evaluation of angiogenic protein platelet-derived endothelial cell growth factor (PD-ECGF) expression in glomeruli. Seckin I, Uzunalan M, Pekpak M, et al. J. Biomed. Sci. 19, 24, (2012)
Dynamics of absolute amount of nephrin in a single podocyte in puromycin aminonucleoside nephrosis rats calculated by quantitative glomerular proteomics approach with selected reaction monitoring mode. Kawakami H, Kamiie J, Yasuno K, et al. Nephrol. Dial. Transplant. 27(4), 1324-30, (2012)
Induction of podocyte-derived VEGF ameliorates podocyte injury and subsequent abnormal glomerular development caused by puromycin aminonucleoside. Ma J, Matsusaka T, Yang HC, et al. Pediatr. Res. 70(1), 83-9, (2011)
Expression and localization of insulin-like growth factor binding proteins in normal and proteinuric kidney glomeruli. Fujinaka H, Katsuyama K, Yamamoto K, et al. Nephrology (Carlton.) 15(7), 700-9, (2010)
[Differential expressions of miRNAs in kidney in puromycin aminonucleoside nephropathy model and intervened effects of Leizhi capsule]. Sun W, Li CQ, Chen JH, et al. Zhongguo Zhong Yao Za Zhi 38(1), 81-90, (2013)
Podocyte Wnt/ß-catenin pathway is activated by integrin-linked kinase in clinical and experimental focal segmental glomerulosclerosis. Naves MA, Requião-Moura LR, Soares MF, et al. J. Nephrol. 25(3), 401-9, (2012)
Connective tissue growth factor modulates podocyte actin cytoskeleton and extracellular matrix synthesis and is induced in podocytes upon injury. Fuchshofer R, Ullmann S, Zeilbeck LF, et al. Histochem. Cell Biol. 136(3), 301-19, (2011)
Marker expression, behaviors, and responses vary in different lines of conditionally immortalized cultured podocytes. Chittiprol S, Chen P, Petrovic-Djergovic D, et al. Am. J. Physiol. Renal Physiol. 301(3), F660-71, (2011)
Silver-enhanced immunogold scanning electron microscopy using vibratome sections of rat kidneys: detection of albumin filtration and reabsorption. Kinugasa S, Tojo A, Sakai T, et al. Med. Mol. Morphol. 43(4), 218-25, (2010)
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