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T2449

Sigma-Aldrich

Triglyceride Reagent

used for quantitative enzymatic determination of triglyerides

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About This Item

UNSPSC Code:
12352200
NACRES:
NA.25

Quality Level

usage

10 mL sufficient for 50 tests

storage temp.

2-8°C

Application

The triglyceride and free glycerol kits are for the quantitative determination of glycerol, total triglycerides or free triglycerides.

Principle

The triglyceride reagent includes lipase for hydrolysis of triglycerides to glycerol.

Linkage

In addition to kits, the individual reagents and glycerol standard are available separately when fewer reactions are needed.

related product

Product No.
Description
Pricing

required but not provided

standard

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Personal Protective Equipment

dust mask type N95 (US), Eyeshields, Gloves

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Even Fjære et al.
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To understand the consequences of the complete elimination of E2F regulation, we profiled the proteome of Drosophila dDP mutants that lack functional E2F/DP complexes. The results uncovered changes in the larval fat body, a differentiated tissue that grows via endocycles.
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In adipocytes, lipid droplet (LD) size reflects a balance of triglyceride synthesis (lipogenesis) and hydrolysis (lipolysis). Perilipin A (Peri A) is the most abundant phosphoprotein on the surface of adipocyte LDs and has a crucial role in lipid storage and
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Insects are known to respond to seasonal and adverse environmental changes by entering dormancy, also known as diapause. In some insect species, including
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Glycogen synthase kinase (GSK)-3beta plays an important role in cardiomyopathies. Cardiac-specific metallothionein-overexpressing transgenic (MT-TG) mice were highly resistant to diabetes-induced cardiomyopathy. Therefore, we investigated whether metallothionein cardiac protection against diabetes is mediated by inactivation of GSK-3beta. Diabetes was induced with

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