Fusion protein amino acids 1-389 (extracellular N-terminus) of rat GluA1/GluR1
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PBS pH 7.4, 50% glycerol, 0.1% sodium azide
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PRRT2 loss-of-function mutations have been associated with familial paroxysmal kinesigenic dyskinesia (PKD), infantile convulsions and choreoathetosis, and benign familial infantile seizures. Dystonia is the foremost involuntary movement disorder manifest by patients with PKD. Using a lacZ reporter and quantitative reverse-transcriptase
The Journal of biological chemistry, 297(2), 100949-100949 (2021-07-13)
Long-term potentiation (LTP) and long-term depression (LTD) of excitatory neurotransmission are believed to be the neuronal basis of learning and memory. Both processes are primarily mediated by neuronal activity-induced transport of postsynaptic AMPA-type glutamate receptors (AMPARs). While AMPAR subunits and
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