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719099

Sigma-Aldrich

Acetaldehyde solution

5 M in THF

Linear Formula:
CH3CHO
CAS Number:
Molecular Weight:
44.05
MDL number:
eCl@ss:
39021102
PubChem Substance ID:
NACRES:
NA.22

form

liquid

Quality Level

concentration

5 M in THF

refractive index

n20/D 1.389

density

0.868 g/mL at 20 °C
0.843 g/mL at 25 °C

storage temp.

2-8°C

SMILES string

[H]C(C)=O

InChI

1S/C2H4O/c1-2-3/h2H,1H3

InChI key

IKHGUXGNUITLKF-UHFFFAOYSA-N

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Related Categories

Signal Word

Danger

Hazard Classifications

Carc. 1B - Eye Irrit. 2 - Flam. Liq. 2 - Muta. 2

Supplementary Hazards

Storage Class Code

3 - Flammable liquids

WGK

WGK 3

Flash Point(F)

-25.1 °F - closed cup

Flash Point(C)

-31.7 °C - closed cup

Certificate of Analysis

Certificate of Origin

Nikoline Borgermann et al.
The EMBO journal, 38(8) (2019-03-28)
DNA-protein crosslinks (DPCs) are highly cytotoxic lesions that obstruct essential DNA transactions and whose resolution is critical for cell and organismal fitness. However, the mechanisms by which cells respond to and overcome DPCs remain incompletely understood. Recent studies unveiled a
Hyo-Jung Kwon et al.
Hepatology (Baltimore, Md.), 60(1), 146-157 (2014-02-05)
Aldehyde dehydrogenase 2 (ALDH2) is the major enzyme that metabolizes acetaldehyde produced from alcohol metabolism. Approximately 40-50% of East Asians carry an inactive ALDH2 gene and exhibit acetaldehyde accumulation after alcohol consumption. However, the role of ALDH2 deficiency in the
Mashiko Setshedi et al.
Oxidative medicine and cellular longevity, 3(3), 178-185 (2010-08-19)
Chronic alcohol abuse causes liver disease that progresses from simple steatosis through stages of steatohepatitis, fibrosis, cirrhosis, and eventually hepatic failure. In addition, chronic alcoholic liver disease (ALD), with or without cirrhosis, increases risk for hepatocellular carcinoma (HCC). Acetaldehyde, a
Tommaso Mello et al.
Molecular aspects of medicine, 29(1-2), 17-21 (2008-01-01)
Alcohol abuse is one of the major causes of liver fibrosis worldwide. Although the pathogenesis of liver fibrosis is a very complex phenomenon involving different molecular and biological mechanisms, several lines of evidence established that the first ethanol metabolite, acetaldehyde
Mikko Salaspuro
Journal of digestive diseases, 12(2), 51-59 (2011-03-16)
Aldehyde dehydrogenase (ALDH2) and alcohol dehydrogenase (ADH) gene polymorphisms associating with enhanced acetaldehyde exposure and markedly increased cancer risk in alcohol drinkers provide undisputable evidence for acetaldehyde being a local carcinogen not only in esophageal but also in gastric cancer.

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