Showing 1-8 of 8 résultats for "MAB1617"
Minako Tateno et al.
Human molecular genetics, 18(5), 942-955 (2008-12-18)
Mutations in the superoxide dismutase 1 (sod1) gene cause familial amyotrophic lateral sclerosis (FALS), likely due to the toxic properties of misfolded mutant SOD1 protein. Here we demonstrated that, starting from the pre-onset stage of FALS, misfolded SOD1 species associates...
Ping Shi et al.
Biochimica et biophysica acta, 1802(9), 707-716 (2010-06-01)
Transport of material and signals between extensive neuronal processes and the cell body is essential to neuronal physiology and survival. Slowing of axonal transport has been shown to occur before the onset of symptoms in amyotrophic lateral sclerosis (ALS). We...
Monoclonal antibodies to kinesin heavy and light chains stain vesicle-like structures, but not microtubules, in cultured cells.
Pfister, K K, et al.
The Journal of cell biology, 108, 1453-1463 (1989)
Visualization and biochemical analyses of the emerging mammalian 14-3-3-phosphoproteome.
Johnson, C; Tinti, M; Wood, NT; Campbell, DG; Toth, R; Dubois, F; Geraghty, KM; Wong et al.
Molecular and Cellular Proteomics null
Submolecular domains of bovine brain kinesin identified by electron microscopy and monoclonal antibody decoration.
Hirokawa, N, et al.
Cell, 56, 867-878 (1989)
Anuttama Kulkarni et al.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 31(3), 965-974 (2016-12-07)
Acetylcholinesterase (AChE), which is implicated in the pathophysiology of neurological disorders, is distributed along the axon and enriched at the presynaptic basal lamina. It hydrolyses the neurotransmitter acetylcholine, which inhibits synaptic transmission. Aberrant AChE activity and ectopic axonal accumulation of...
Fast axonal transport of kinesin in the rat visual system: functionality of kinesin heavy chain isoforms.
Elluru, R G, et al.
Molecular Biology of the Cell, 6, 21-40 (1995)

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