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SAB1304615

Sigma-Aldrich

ANTI-NOX4 (N-TERM) antibody produced in rabbit

IgG fraction of antiserum, buffered aqueous solution

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Synonym(s):
NADPH oxidase 4, RENOX
NACRES:
NA.41

biological source

rabbit

Quality Level

conjugate

unconjugated

antibody form

IgG fraction of antiserum

antibody product type

primary antibodies

clone

polyclonal

form

buffered aqueous solution

mol wt

66932 Da

species reactivity

human

technique(s)

western blot: 1:1000

NCBI accession no.

UniProt accession no.

shipped in

wet ice

storage temp.

−20°C

target post-translational modification

unmodified

Gene Information

human ... NOX4(50507)

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This Item
ABC459SAB5700950SAB4503153
Anti-NOX4 Antibody from rabbit

ABC459

Anti-NOX4 Antibody

Quality Level

100

Quality Level

100

Quality Level

100

Quality Level

100

antibody form

IgG fraction of antiserum

antibody form

purified antibody

antibody form

-

antibody form

affinity isolated antibody

conjugate

unconjugated

conjugate

-

conjugate

-

conjugate

unconjugated

mol wt

66932 Da

mol wt

-

mol wt

-

mol wt

antigen 86 kDa

UniProt accession no.

Q9NPH5

UniProt accession no.

Q9NPH5

UniProt accession no.

Q9NPH5

UniProt accession no.

Q96PH1

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Physical form

Purified polyclonal antibody supplied in PBS with 0.09% (W/V) sodium azide. This antibody is purified through a protein A column, followed by peptide affinity purification.

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Storage Class Code

10 - Combustible liquids

WGK

nwg

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


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Elis Aguiar Morra et al.
Free radical biology & medicine, 130, 174-188 (2018-10-14)
We previously demonstrated that the loss of female hormones induces cardiac and mitochondrial dysfunction in the female heart. Here, we show the impact of endurance training for twelve weeks, a nonpharmacological therapy against cardiovascular disease caused by ovariectomy and its

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