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30968

Sigma-Aldrich

Sodium deoxycholate monohydrate

BioUltra, ≥99.0% (NT)

Sinónimos:
Desoxycholic acid sodium salt, 3α,12α-Dihydroxy-5β-cholanic acid sodium salt, 7-Deoxycholic acid sodium salt
Empirical Formula (Hill Notation):
C24H39NaO4 · H2O
Número de CAS:
Peso molecular:
432.57
Beilstein:
3643164
Número de EC:
Número MDL:
ID de la sustancia en PubChem:

Nivel de calidad

300

descripción

anionic

línea de producto

BioUltra

ensayo

≥99.0% (NT)

actividad óptica

[α]20/D +44±2°, c = 2% in H2O

mol peso

average mol wt 1200-5000

número de agregación

3-12

impurezas

insoluble matter, passes filter test
≤0.5% cholic acid (TLC)
≤6.1% water

pH

7.5-9.5 (25 °C, 0.1 M in H2O)

CMC

2-6

solubilidad

H2O: 0.1 M at 20 °C, clear, colorless

trazas de anión

chloride (Cl-): ≤50 mg/kg
sulfate (SO42-): ≤200 mg/kg

trazas de catión

Al: ≤20 mg/kg
Ba: ≤5 mg/kg
Bi: ≤5 mg/kg
Cd: ≤5 mg/kg
Co: ≤5 mg/kg
Cr: ≤5 mg/kg
Cu: ≤5 mg/kg
Fe: ≤5 mg/kg
Li: ≤5 mg/kg
Mg: ≤20 mg/kg
Mn: ≤5 mg/kg
Mo: ≤5 mg/kg
Ni: ≤5 mg/kg
Pb: ≤5 mg/kg
Sr: ≤20 mg/kg
Zn: ≤5 mg/kg

λ

0.1 M in H2O

Absorción UV

λ: 260 nm Amax: 0.10
λ: 280 nm Amax: 0.08

HLB

16

SMILES string

O.[Na+].C[C@H](CCC([O-])=O)C1CCC2C3CCC4C[C@H](O)CC[C@]4(C)C3C[C@H](O)[C@]12C

InChI

1S/C24H40O4.Na.H2O/c1-14(4-9-22(27)28)18-7-8-19-17-6-5-15-12-16(25)10-11-23(15,2)20(17)13-21(26)24(18,19)3;;/h14-21,25-26H,4-13H2,1-3H3,(H,27,28);;1H2/q;+1;/p-1/t14-,15-,16-,17+,18-,19+,20+,21+,23+,24-;;/m1../s1

InChI key

NDVHNZISGVSFMP-WTCAICSISA-M

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Descripción general

Sodium deoxycholate monohydrate is a lipid bile salt used in drug delivery systems.

Aplicación

Powerful solubilization agent; used in the solubilization of estrogen synthetase from human placental microsomes.

Acciones bioquímicas o fisiológicas

Bile salt

pictogramas

Exclamation mark

Palabra de señalización

Warning

Frases de peligro

Consejos de prudencia

Clasificaciones de peligro

Acute Tox. 4 Oral

Código de clase de almacenamiento

13 - Non Combustible Solids

WGK

WGK 3

Punto de inflamabilidad F

Not applicable

Punto de inflamabilidad C

Not applicable

Equipo de protección personal

dust mask type N95 (US), Eyeshields, Gloves

Certificado de Análisis

Certificado de origen

Suguru Yamasaki et al.
Scientific reports, 9(1), 177-177 (2019-01-19)
During infection, Salmonella senses and responds to harsh environments within the host. Persistence in a bile-rich environment is important for Salmonella to infect the small intestine or gallbladder and the multidrug efflux system AcrAB-TolC is required for bile resistance. The
Richard R Sprenger et al.
Cell reports, 34(5), 108710-108710 (2021-02-04)
Diurnal regulation of whole-body lipid metabolism plays a vital role in metabolic health. Although changes in lipid levels across the diurnal cycle have been investigated, the system-wide molecular responses to both short-acting fasting-feeding transitions and longer-timescale circadian rhythms have not
Fernando Martínez-Montañés et al.
Cell reports, 32(6), 108024-108024 (2020-08-14)
The ability to remodel lipid metabolism under changing conditions is pivotal for cellular functionality and homeostasis. Here, we characterize the regulatory landscape of phosphorylation-based signaling events across the life cycle of Saccharomyces cerevisiae and determine its impact on the regulation
Arvind Venkat Namuduri et al.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 34(2), 2269-2286 (2020-01-08)
SUMOylation is a dynamic, reversible, enzymatic drug-targetable post-translational modification (PTM) reaction where the Small Ubiquitin-like Modifier (SUMO) moieties are attached to proteins. This reaction regulates various biological functions like cell growth, differentiation, and it is crucial for maintaining organ homeostasis.
Xiuxiu Liu et al.
Experimental cell research, 395(2), 112234-112234 (2020-08-22)
Skeletal muscle preservation is a dynamic process that involves constant repair and regeneration. However, the regenerative capacity of muscle cells declines in hyperglycemia. This study aimed to explore the molecular mechanisms underlying this glucotoxicity during myoblast differentiation. C2C12 cells were exposed

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