Przemyslaw Swiatkowski et al.
Biochemical and biophysical research communications, 433(3), 354-357 (2013-03-21)
In the pharynx of Caenorhabditis elegans, the accessory subunit MPS-4, homolog to human KCNE1, forms a complex with K(+) channel EXP-2 that terminates the action potential. An aspartate residue critical for KCNE1 function, asp76, is conserved in MPS-4 (asp74). Here