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Palabra clave:'sab4200483'
Mostrando 1-10 of 10 resultados para "sab4200483" en el plazo de Papeles
The two faces of Janus kinases and their respective STATs in mammary gland development and cancer
Wagner KU and Schmidt JW
Journal of Carcinogenesis, 10, 273-287 (2011)
Zhiyong Zhang et al.
Environmental toxicology, 36(8), 1557-1566 (2021-05-07)
Delphinidin is an anthocyanin that belongs to the group of flavonoids that exert numerous biological activities. However, the molecular mechanisms underlying the anticancer effects of delphinidin remain poorly understood. In our study we analyzed delphinidin modulate STAT-3 and MAPKinase signaling
Targeting growth hormone receptor in human melanoma cells attenuates tumor progression and epithelial mesenchymal transition via suppression of multiple oncogenic pathways
Basu R, et al.
Oncotarget, 8(13), 21579-21579 (2017)
Spleen deflation and beyond: The pros and cons of Janus kinase 2 inhibitor therapy for patients with myeloproliferative neoplasms
Quintas CA and Verstovsek S
Cancer, 118(4), 870-877 (2012)
Janus kinases in immune cell signaling
Ghoreschi K, et al.
Immunological Reviews, 228(1), 273-287 (2009)
Thymoquinone inhibits proliferation, induces apoptosis and chemosensitizes human multiple myeloma cells through suppression of signal transducer and activator of transcription 3 activation pathway
Li F, et al.
British Journal of Pharmacology, 161(3), 541-554 (2010)
Reetobrata Basu et al.
Oncotarget, 8(13), 21579-21598 (2017-02-23)
Recent reports have confirmed highest levels of growth hormone (GH) receptor (GHR) transcripts in melanoma, one of the most aggressive forms of human cancer. Yet the mechanism of GH action in melanoma remains mostly unknown. Here, using human malignant melanoma
Chengliang Yuan et al.
Journal of biochemical and molecular toxicology, 36(1), e22943-e22943 (2021-11-02)
Eriocitrin, a lemons flavanone, exhibits several biological properties, antiproliferative, and proapoptotic effects. However, its molecular mechanical action is not entirely clarified. Oxidative stress causes abnormal stimulation of signal transducer and activator of transcription 3 (STAT3) and c-Jun NH2-terminal kinase (JNK), p38
The JAK/STAT signaling pathway
Rawlings JS
Journal of Cell Science, 117(8), 1281-1283 (2004)
Cross talk of signals between EGFR and IL-6R through JAK2/STAT3 mediate epithelial-mesenchymal transition in ovarian carcinomas
Colomiere M, et al.
British Journal of Cancer, 100(1), 134-134 (2009)
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