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SAB4200489

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Palabra clave:'SAB4200489'
Mostrando 1-7 of 7 resultados para "SAB4200489" en el plazo de Papeles
Claudin-1 and -2: Novel Integral Membrane Proteins Localizing at Tight Junctions with No Sequence Similarity to Occludin
The Journal of Cell Biology, 141(7) (1998)
Wanghui Jing et al.
American journal of translational research, 10(7), 2137-2147 (2018-08-11)
Chronic kidney disease (CKD) results in various central nervous systems (CNS) disorders including cognitive dysfunction, depression, anxiety, movement disorders, seizures and encephalopathy. Uremic retention products, oxidative stress, inflammation and impaired blood-brain barrier have been implicated as the major mediators of
The Tight Junction Protein, Occludin, Regulates the Directional Migration of Epithelial Cells
Dan Du, et al.
Developmental Cell (2010)
Recessive Mutations in the Gene Encoding the Tight Junction Protein Occludin Cause Band-like Calcification with Simplified Gyration and Polymicrogyria
Mary C, et al.
American Journal of Human Genetics (2010)
Splicing Diversity of the Human OCLN Gene and Its Biological Significance for Hepatitis C Virus Entry? ?
Indu Kohaar, et al.
Molecular and Cellular Biology (2010)
Analyses of Gingival Adhesion Molecules in Periodontitis: Theoretical In Silico, Comparative In Vivo, and Explanatory In Vitro Models
Ulvi K, et al.
Journal of Periodontal Research (2015)
Complex Phenotype of Mice Lacking Occludin, a Component of Tight Junction Strands
Complex Phenotype of Mice Lacking Occludin, a Component of Tight Junction Strands
Mitinouri Saitou, et al
Molecular Biology of the Cell (2017)
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