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Solketal methacrylate

50 wt. % in dichloromethane, contains ~280 ppm 4-tert-butylcatechol as inhibitor

Glycerine-1-methacrylate acetone ketal, SMA, GMAK, (2,2-Dimethyl-1,3-dioxolan-4-yl)methyl methacrylate
Empirical Formula (Hill Notation):
CAS Number:
Molecular Weight:
MDL number:
PubChem Substance ID:

Quality Level


~280 ppm 4-tert-butylcatechol as inhibitor


50 wt. % in dichloromethane

refractive index

n20/D 1.438


1.153 g/mL at 25 °C

storage temp.


SMILES string




InChI key


Signal Word


Hazard Classifications

Carc. 2 - Eye Dam. 1 - Flam. Liq. 3 - Skin Irrit. 2

Storage Class Code

3 - Flammable liquids



Flash Point(F)

81.0 °F

Flash Point(C)

27.2 °C

Certificate of Analysis

Certificate of Origin

Sarah L O'Beirne et al.
Journal of immunology (Baltimore, Md. : 1950), 195(6), 2788-2796 (2015-08-14)
Epithelial to mesenchymal cell transition (EMT), whereby fully differentiated epithelial cells transition to a mesenchymal phenotype, has been implicated in the pathogenesis of idiopathic pulmonary fibrosis (IPF). CXCR3 and its ligands are recognized to play a protective role in pulmonary
Caroline J Zeiss et al.
G3 (Bethesda, Md.), 9(8), 2637-2646 (2019-07-03)
Anthracyclines cause progressive cardiotoxicity whose ultimate severity is individual to the patient. Genetic determinants contributing to this variation are difficult to study using current mouse models. Our objective was to determine whether a spectrum of anthracycline induced cardiac disease can
Rui Zheng et al.
The Journal of investigative dermatology, 139(9), 2029-2038 (2019-03-18)
After cutaneous injury, keratinocytes secrete paracrine factors that regulate wound cell functions; dysregulation of this signaling can lead to wound pathologies. Previously, we established that keratinocyte integrin α3β1 promotes wound angiogenesis through paracrine stimulation of endothelial cells. We hypothesize here
Frank Y Ma et al.
Nephrology (Carlton, Vic.), 24(9), 983-991 (2019-07-18)
Protease-activated receptor 2 (PAR2) has been implicated in the development of renal inflammation and fibrosis. In particular, activation of PAR2 in cultured tubular epithelial cells induces extracellular signal-regulated kinase signalling and secretion of fibronectin, C-C Motif Chemokine Ligand 2 (CCL2)
Hiroyasu Okazaki et al.
Experimental lung research, 45(7), 188-199 (2019-07-13)
Purpose/Aim of the Study: Wnt/β-catenin signaling was reported to be activated in pulmonary fibrosis, and was focused on as a target for antifibrotic therapy. However, the mechanism how the inhibition of Wnt/β-catenin signaling ameliorate pulmonary fibrosis has not been fully

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