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Detection of bacterial pyrogens on the basis of their effects on gamma interferon-mediated formation of neopterin or nitrite in cultured monocyte cell lines.

G Werner-Felmayer et al.

Clinical and diagnostic laboratory immunology, 2(3), 307-313 (1995-05-01)

In a number of mammalian cell types, pteridine biosynthesis from guanosine 5'-triphosphate and formation of nitric oxide from L-arginine are induced by gamma interferon (IFN-gamma) and bacterial lipopolysaccharide (LPS). We assessed the possibility of using such metabolic alterations for the

Binding of different forms of lipopolysaccharide and gene expression in bovine blood neutrophils.

M Worku et al.

Journal of dairy science, 92(7), 3185-3193 (2009-06-17)

The objective of this study was to evaluate the effect of smooth (S) and rough (R) forms of lipopolysaccharide (LPS) on gene expression in bovine blood neutrophils. Isolated neutrophils (10(7) cells/mL) were treated with Escherichia coli LPS serotype O111:B4 (S+R)

Molecular Interactions of Lipopolysaccharide with an Outer Membrane Protein from Pseudomonas aeruginosa Probed by Solution NMR.

Iga Kucharska et al.

Biochemistry, 55(36), 5061-5072 (2016-08-18)

Pseudomonas aeruginosa is an opportunistic human pathogen causing pneumonias that are particularly severe in cystic fibrosis and immunocompromised patients. The outer membrane (OM) of P. aeruginosa is much less permeable to nutrients and other chemical compounds than that of Escherichia

TNFα alters occludin and cerebral endothelial permeability: Role of p38MAPK.

Yawen Ni et al.

PloS one, 12(2), e0170346-e0170346 (2017-02-09)

Occludin is a key tight junction (TJ) protein in cerebral endothelial cells (CECs) playing an important role in modulating blood-brain barrier (BBB) functions. This protein (65kDa) has been shown to engage in many signaling pathways and phosphorylation by both tyrosine

Mutually constructive roles of Ail and LPS in Yersinia pestis serum survival.

Chandan Singh et al.

Molecular microbiology, 114(3), 510-520 (2020-05-29)

The outer membrane is a key virulence determinant of gram-negative bacteria. In Yersinia pestis, the deadly agent that causes plague, the protein Ail and lipopolysaccharide (LPS)6 enhance lethality by promoting resistance to human innate immunity and antibiotics, enabling bacteria to

Rough-Form Lipopolysaccharide Increases Apoptosis in Human CD4⁺ and CD8⁺ T Lymphocytes.

J S Nielsen et al.

Scandinavian journal of immunology, 75(2), 193-202 (2011-08-23)

Immunosuppression induced by lymphocyte apoptosis is considered an important factor in the pathogenesis of sepsis and has been demonstrated in both animal models of lipopolysaccharide (LPS)-induced endotoxemia and septic patients. As rough-form LPS (R-LPS) has recently been shown to elicit

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