Harry Karmouty-Quintana et al.
FASEB journal : official publication of the Federation of American Societies for Experimental Biology, 29(1), 50-60 (2014-10-17)
Idiopathic pulmonary fibrosis (IPF) is a lethal, fibroproliferative disease. Pulmonary hypertension (PH) can develop secondary to IPF and increase mortality. Alternatively, activated macrophages (AAMs) contribute to the pathogenesis of both IPF and PH. Here we hypothesized that adenosine signaling through