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779423

Sigma-Aldrich

Ammonia solution

4 M in methanol

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Linear Formula:
NH3
CAS Number:
Molecular Weight:
17.03
MDL number:
PubChem Substance ID:
NACRES:
NA.21

form

liquid

concentration

3.6-4.4 M (back titration with NaOH)
3400-4400 mmol/L (back titration with NaOH)
4 M in methanol

SMILES string

N

InChI

1S/H3N/h1H3

InChI key

QGZKDVFQNNGYKY-UHFFFAOYSA-N

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1 of 4

This Item
341428392693407666
Ammonia solution 4 M in methanol

Sigma-Aldrich

779423

Ammonia solution

Ammonia solution 2.0 M in methanol

Sigma-Aldrich

341428

Ammonia solution

Ammonia solution 2.0 M in isopropanol

Sigma-Aldrich

392693

Ammonia solution

Ammonia solution 0.4 M in dioxane

Sigma-Aldrich

407666

Ammonia solution

concentration

3.6-4.4 M (back titration with NaOH), 3400-4400 mmol/L (back titration with NaOH), 4 M in methanol

concentration

1.90-2.20 M (with NaOH, titration), 2.0 M in methanol

concentration

2.0 M in isopropanol, 2.0-2.2 M (by NaOH, titration)

concentration

0.4 M in dioxane, 450-600 mmol/L (titration)

Quality Level

200

Quality Level

200

Quality Level

100

Quality Level

100

General description

Ammonia solution (ammonia in methanol) is a colorless gas with a characteristic strong odor. It is highly soluble in water, alcohol, chloroform ether, ethanol, and methanol. Ammonia solution is commonly used as a base.

Signal Word

Danger

Hazard Classifications

Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral - Aquatic Chronic 3 - Eye Dam. 1 - Flam. Liq. 2 - Skin Corr. 1B - STOT SE 1

Target Organs

Eyes

Supplementary Hazards

Storage Class Code

3 - Flammable liquids

WGK

WGK 2

Flash Point(F)

57.2 °F - open cup

Flash Point(C)

14 °C - open cup


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Eagleson M.
Concise Encyclopedia Chemistry, 65-66 (1994)
Ledgard J.
The Preparatory Manual of Explosives., 41-41 (2007)
Marion Vallet et al.
Kidney international, 88(1), 137-145 (2015-03-12)
Recent studies suggest that alkalinizing treatments improve the course of chronic kidney disease (CKD), even in patients without overt metabolic acidosis. Here, we tested whether a decreased ability in excreting urinary acid rather than overt metabolic acidosis may be deleterious
Olivier Braissant et al.
Journal of inherited metabolic disease, 36(4), 595-612 (2012-10-31)
Hyperammonemia can be caused by various acquired or inherited disorders such as urea cycle defects. The brain is much more susceptible to the deleterious effects of ammonium in childhood than in adulthood. Hyperammonemia provokes irreversible damage to the developing central
Lars Nybo et al.
The Journal of physiology, 563(Pt 1), 285-290 (2004-12-22)
We evaluated whether peripheral ammonia production during prolonged exercise enhances the uptake and subsequent accumulation of ammonia within the brain. Two studies determined the cerebral uptake of ammonia (arterial and jugular venous blood sampling combined with Kety-Schmidt-determined cerebral blood flow;

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