MAB045P

Anti-Fluorescein (FITC) Antibody, clone 5D6.2, Peroxidase conjugated

Name: Anti-Fluorescein (FITC) Antibody, clone 5D6.2, Peroxidase conjugated
Brand: MM
Price: 372 USD

clone 5D6.2, Chemicon^®, from mouse

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About This Item

UNSPSC Code:

12352203

eCl@ss:

32160702

NACRES:

NA.41

Key Documents

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biological source

mouse

Quality Level

100

conjugate

peroxidase conjugate

antibody form

purified immunoglobulin

antibody product type

primary antibodies

clone

5D6.2, monoclonal

species reactivity (predicted by homology)

all

manufacturer/tradename

Chemicon^®

technique(s)

ELISA: suitable
immunohistochemistry: suitable

isotype

IgG2a

shipped in

wet ice

target post-translational modification

unmodified

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This Item	MAB045	CBL458F	SAB5200235
Sigma-Aldrich MAB045P Anti-Fluorescein (FITC) Antibody, clone 5D6.2, Peroxidase conjugated	Sigma-Aldrich MAB045 Anti-Fluorescein (FITC) Antibody, clone 5D6.2	Sigma-Aldrich CBL458F Anti-CD49f Antibody, FITC conjugated, clone 4F10, FITC conjugated	Sigma-Aldrich SAB5200235 Monoclonal Anti-Kcc2 - Hrp antibody produced in mouse
clone 5D6.2, monoclonal	clone 5D6.2, monoclonal	clone 4F10, monoclonal	clone S1-12, monoclonal
biological source mouse	biological source mouse	biological source mouse	biological source mouse
antibody form purified immunoglobulin	antibody form purified immunoglobulin	antibody form purified immunoglobulin	antibody form purified immunoglobulin
conjugate peroxidase conjugate	conjugate -	conjugate FITC conjugate	conjugate peroxidase conjugate
isotype IgG2a	isotype IgG2a	isotype IgG2b	isotype IgG2a
shipped in wet ice	shipped in wet ice	shipped in wet ice	shipped in wet ice

Application

Detect Fluorescein (FITC) using this Anti-Fluorescein (FITC) Antibody, clone 5D6.2, Peroxidase conjugated validated for use in ELISA, IH.

ELISA

Immunohistochemistry

Optimal working dilutions must be determined by end user.

Biochem/physiol Actions

Fluorescein (FITC)

Physical form

0.005M PBS, 50% Stabilzyme, pH7.1

Preparation Note

Maintain at +2-8°C for up to 6 months.

Other Notes

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

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pictograms

GHS07

signalword

Warning

hcodes

H315,H317,H319,H412

pcodes

P261 - P264 - P273 - P280 - P302 + P352 - P305 + P351 + P338

Hazard Classifications

Aquatic Chronic 3 - Eye Irrit. 2 - Skin Irrit. 2 - Skin Sens. 1

Storage Class

11 - Combustible Solids

wgk_germany

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Tumor necrosis factor-α induces increased lung vascular permeability: a role for GSK3α/β.

Amy Barton-Pai et al.

European journal of pharmacology, 657(1-3), 159-166 (2011-02-15)

We tested the hypothesis that glycogen synthase kinase 3α/β (GSK3α/β) modulates tumor necrosis factor-a (TNF) induced increased lung vascular permeability. Rats were treated with TNF (i.v., ~100ng/ml) or vehicle 0.5h, 4.0h and 24.0h prior to lung isolation. Rats were co-treated

Endothelial Wnt/β-catenin signaling inhibits glioma angiogenesis and normalizes tumor blood vessels by inducing PDGF-B expression.

Marco Reis et al.

The Journal of experimental medicine, 209(9), 1611-1627 (2012-08-22)

Endothelial Wnt/β-catenin signaling is necessary for angiogenesis of the central nervous system and blood-brain barrier (BBB) differentiation, but its relevance for glioma vascularization is unknown. In this study, we show that doxycycline-dependent Wnt1 expression in subcutaneous and intracranial mouse glioma

Stress-resistant Translation of Cathepsin L mRNA in Breast Cancer Progression.

Martina Tholen et al.

The Journal of biological chemistry, 290(25), 15758-15769 (2015-05-10)

The cysteine protease cathepsin L (CTSL) is often thought to act as a tumor promoter by enhancing tumor progression and metastasis. This goes along with increased CTSL activity in various tumor entities; however, the mechanisms leading to high CTSL levels

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