MAB413

Sigma-Aldrich

Anti-Interferon-α Antibody, clone ST126

ascites fluid, clone ST126, Chemicon®

eCl@ss:
32160702
NACRES:
NA.41
Pricing and availability is not currently available.

Quality Level

biological source

mouse

antibody form

ascites fluid

clone

ST126, monoclonal

species reactivity

human

mfr. no.

Chemicon®

application(s)

neutralization: suitable

isotype

IgG2b

NCBI accession no.

UniProt accession no.

shipped in

dry ice

Specificity

Human interferon-alpha sub-species. Distinguishes between sub-species of human alpha-interferon. Can be used to detect interferon oligomers.

Immunogen

Human alpha-interferon

Application

Anti-Interferon-α Antibody, clone ST126 detects level of Interferon-α & has been published & validated for use in NEUT.
Research Sub Category
Cytokines & Cytokine Receptors
Neutralizes anti-viral activity of interferon.

Dilute with buffer pH 7.5 - 8.0 to desired working volumes. Final working dilutions must be determined by end user. For extensive dilution use protein-containing or other stabilizing medium.
Research Category
Inflammation & Immunology

Physical form

Liquid, no preservatives.

Storage and Stability

Maintain at -20°C for up to 12 months.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

WGK Germany

WGK 1

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Monoclonal antibodies that distinguish between subspecies of human interferon-alpha and that detect interferon oligomers.
Shearer, M, et al.
Journal of immunology (Baltimore, Md. : 1950), 133, 3096-3101 (1984)
Marlena M Westcott et al.
Journal of virology, 89(15), 7944-7954 (2015-05-23)
Oncolytic viruses (OV) preferentially kill cancer cells due in part to defects in their antiviral responses upon exposure to type I interferons (IFNs). However, IFN responsiveness of some tumor cells confers resistance to OV treatment. The human type I IFNs...

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