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GE10600002

Amersham Protran® Western blotting membranes, nitrocellulose

pore size 0.45 μm, roll W × L 300 mm × 4 m, pkg of 1 ea

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About This Item

UNSPSC Code:
41105339
NACRES:
NB.22

shelf life

Please be aware this product may be shipped 90 days before the expiration date. For more information on the batch specific expiration date, please contact technical service.

packaging

pkg of 1 ea

manufacturer/tradename

Cytiva 10600002

roll W × L

300 mm × 4 m

pore size

0.45 μm pore size

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General description

Amersham Protran 0.45 NC is a nitrocellulose membrane with a 0.45 µm pore size for use in Western blot protocols. Amersham Protran nitrocellulose membranes offer excellent sensitivity, resolution, and low background noise, especially when used with Amersham ECL, Amersham ECL Prime, or Amersham ECL Select Western Blotting detection reagents. Protran NC membranes are available in popular precut sizes for a wide range of laboratory applications. This product was formerly called Protran BA85.

Application

Amersham Protran® Western blotting membranes, nitrocellulose is suitable for use in:
  • Western blotting
  • Southern blotting
  • Northern blotting

Legal Information

Amersham is a trademark of Cytiva
Protran is a registered trademark of Cytiva

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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Emilia A H Vanni et al.
PLoS pathogens, 16(12), e1009166-e1009166 (2020-12-29)
Herpes simplex virus 1 (HSV-1) infects skin and mucosal epithelial cells and then travels along axons to establish latency in the neurones of sensory ganglia. Although viral gene expression is restricted during latency, the latency-associated transcript (LAT) locus encodes many
Marianna Lucafò et al.
Cancer chemotherapy and pharmacology, 86(3), 361-374 (2020-08-11)
Glucocorticoids (GCs) are commonly used as therapeutic agents for immune-mediated diseases and leukemia. However, considerable inter-individual differences in efficacy have been reported. Several reports indicate that the inhibitor of mTOR rapamycin can reverse GC resistance, but the molecular mechanism involved

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