Anti-Presenilin1 antibody produced in rabbit

affinity isolated antibody, buffered aqueous solution

Anti-Presenilin 1, Anti-PS1, Anti-γ-Secretase subunit presenilin-1, Anti-PSEN1
MDL number:
Pricing and availability is not currently available.

biological source


antibody form

affinity isolated antibody

antibody product type

primary antibodies




buffered aqueous solution

species reactivity

mouse, rat, human


immunofluorescence: suitable
immunohistochemistry: suitable
indirect ELISA: suitable
western blot: suitable



UniProt accession no.

shipped in

dry ice

storage temp.


Gene Information

human ... PSEN1(5663)


a 23 amino acid peptide from near the carboxy-terminus of human presenilin1.


The action of this antibody can be blocked using blocking peptide SBP4203.

Physical form

Solution in phosphate buffered saline containing 0.02% sodium azide


Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.


NONH for all modes of transport

WGK Germany


Flash Point(F)

Not applicable

Flash Point(C)

Not applicable

Certificate of Analysis
Certificate of Origin
Huang Huang et al.
Age (Dordrecht, Netherlands), 38(4), 303-322 (2016-07-22)
Transgenic APPSwe/PS1dE9 (APP/PS1) mice that overproduce amyloid beta (Aβ) are extensively used in the studies of pathogenesis and experimental therapeutics and new drug screening for Alzheimer's disease (AD). However, most of the current literature uses young or adult APP/PS1 mice....
Weixi Feng et al.
Alzheimer's research & therapy, 12(1), 125-125 (2020-10-04)
Soluble beta-amyloid (Aβ) can be cleared from the brain through various mechanisms including enzymatic degradation, glial cell phagocytosis, transport across the blood-brain barrier, and glymphatic clearance. However, the relative contribution of each clearance system and their compensatory effects in delaying...
Min Cao et al.
CNS neuroscience & therapeutics, 24(3), 202-211 (2017-12-24)
Social isolation increases the onset of Alzheimer's disease (AD). Environmental enrichment, a complicated social and physical construct, plays beneficial effects on brain plasticity and function. This study was designed to determine whether physical enrichment can reduce the deleterious consequences of social...

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