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20034

Supelco

ORBO 403 Tenax® TA specially treated (60/80), 100/50 mg

W,W,W separators, O.D. × L 6 mm × 105 mm, pkg of 50 ea

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Synonym(s):
ORBO Tenax® TA

material

W,W,W separators

Agency

NIOSH 2510

product line

ORBO

composition

Bed A, 100 mg
Bed B, 50 mg

packaging

pkg of 50 ea

manufacturer/tradename

ORBO 403

technique(s)

active air sampling: suitable

O.D. × L

6 mm × 105 mm

matrix

Tenax TA (specially treated)

particle size

60-80 mesh

application(s)

air monitoring
environmental
industrial hygiene

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This Item
202582023120051
agency

NIOSH 2510

agency

NIOSH 2544,5506,5515,5800

agency

EPA TO-15, OSHA 52,61

agency

NIOSH 2534,2537

composition

Bed A, 100 mg , Bed B, 50 mg

composition

Bed A, 100 mg , Bed B, 50 mg

composition

Bed A, 150 mg , Bed B, 75 mg

composition

Bed A, 400 mg , Bed B, 200 mg

technique(s)

active air sampling: suitable

technique(s)

active air sampling: suitable

technique(s)

active air sampling: suitable

technique(s)

active air sampling: suitable

product line

ORBO

product line

Amberlite, ORBO

product line

Amberlite, ORBO

product line

Amberlite, ORBO

packaging

pkg of 50 ea

packaging

pkg of 50 ea

packaging

pkg of 25 ea

packaging

pkg of 50 ea

Legal Information

ORBO is a trademark of Sigma-Aldrich Co. LLC
Tenax is a registered trademark of Buchem B.V.

Pictograms

Exclamation mark

Signal Word

Warning

Hazard Statements

Hazard Classifications

Eye Irrit. 2 - Skin Irrit. 2 - STOT SE 3

Target Organs

Respiratory system

Storage Class Code

11 - Combustible Solids

WGK

WGK 3

Flash Point(F)

Not applicable

Flash Point(C)

Not applicable


Certificates of Analysis (COA)

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A Orbo et al.
Gynecologic oncology, 95(1), 82-88 (2004-09-24)
The purpose of the current study was to characterize the role of PTEN in malignant transformation and to evaluate the significance of mutated PTEN exons as prognostic markers in the carcinogenesis of endometrial hyperplasia. A comparison of PTEN mutations as
A Ørbo et al.
BJOG : an international journal of obstetrics and gynaecology, 123(9), 1512-1519 (2015-12-03)
To investigate relapse rates after the successful treatment of patients with non-atypical endometrial hyperplasia who were randomised to either a levonorgestrel-impregnated intrauterine system (LNG-IUS; Mirena(®) ) or two regimens of oral medroxyprogesterone acetate (MPA) after primary histological response. A multicentre
A Orbo et al.
International journal of gynecological pathology : official journal of the International Society of Gynecological Pathologists, 22(2), 141-148 (2003-03-22)
Derangements in the tumor suppressor gene PTEN and the mismatch-repair genes, hMLH1, hMSH2, and hMSH6, have an important role in endometrial carcinogenesis. The purpose of this study was to assess immunohistochemically the pattern of protein expression for these genes in
J P Baak et al.
The American journal of surgical pathology, 25(7), 930-935 (2001-06-23)
Prospective multicenter evaluation of the WHO classification and the morphometric D-score to predict endometrial hyperplasia cancer progression. In 132 endometrial hyperplasias WHO classification was performed by two experienced gynecologic pathologists. The D-score was assessed blindly by technicians in a routine
Anne Ørbo et al.
Anticancer research, 35(12), 6401-6409 (2015-12-08)
To investigate if a levonorgestrel-impregnated intrauterine system (LNG-IUS) was more efficient compared to oral progestin in the clearance of the paired box 2 gene (PAX2) - and phosphatase and tensin homolog (PTEN)-null endometrial glands and assess the significance of PAX2-

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