506-32-1

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Showing 1-8 of 8 results for "506-32-1" within Papers

Factor V leiden and morbid obesity in fatal postoperative pulmonary embolism.

H Blaszyk et al.

Archives of surgery (Chicago, Ill. : 1960), 135(12), 1410-1413 (2000-12-15)

Currently, the risk for postoperative acute pulmonary embolism (APE) is assessed clinically. We hypothesize that the expensive screening for the most common genetic thrombophilic clotting defect (factor V Leiden; R(506)Q) after exclusion of established clinical risk factors does not offer

Cellular signaling underlying atrial tachycardia remodeling of L-type calcium current.

Xiao Yan Qi et al.

Circulation research, 103(8), 845-854 (2008-08-30)

Atrial tachycardia (AT) downregulates L-type Ca(2+) current (I(CaL)) and causes atrial fibrillation-promoting electric remodeling. This study assessed potential underlying signal transduction. Cultured adult canine atrial cardiomyocytes were paced at 0, 1, or 3 Hz (P0, P1, P3) for up to

Molecular characterization of the promoter region of a neuroendocrine tumor marker, IA-1.

Q Li et al.

Biochemical and biophysical research communications, 236(3), 776-781 (1997-07-30)

IA-1 is an intronless gene, which encodes a 510 amino acid protein with a zinc-finger DNA-binding motif that is expressed in tumors of neuroendocrine origin. The 5'-upstream region of the IA-1 gene was recently sequenced. In this paper, the regulatory

Reconstitution of ancestral green visual pigments of zebrafish and molecular mechanism of their spectral differentiation.

Akito Chinen et al.

Molecular biology and evolution, 22(4), 1001-1010 (2005-01-14)

We previously reported that zebrafish have four tandemly duplicated green (RH2) opsin genes (RH2-1, RH2-2, RH2-3, and RH2-4). Absorption spectra vary widely among the four photopigments reconstituted with 11-cis retinal, with their peak absorption spectra (lambda(max)) being 467, 476, 488

Molecular cloning and functional analysis of the promoter of rat skeletal muscle voltage-sensitive sodium channel subtype 2 (rSkM2): evidence for muscle-specific nuclear protein binding to the core promoter.

Z H Sheng et al.

DNA and cell biology, 13(1), 9-23 (1994-01-01)

rSkM2 is a tetrodotoxin-resistant rat skeletal muscle voltage-sensitive sodium channel that is expressed in immature and denervated skeletal muscle and in adult heart. We have isolated a 3.7-kb gene segment that contains the first exon, multiple transcription initiation sites, the

Selected gene polymorphisms and their interaction with maternal smoking, as risk factors for gastroschisis.

Claudine P Torfs et al.

Birth defects research. Part A, Clinical and molecular teratology, 76(10), 723-730 (2006-10-20)

Gastroschisis is a severe birth defect in which the infant is born with a portion of the intestines extruding through a small tear in the abdominal wall, usually to the right of the umbilical cord. Its etiology is unknown, but

CYP2C19 polymorphism affects single-dose pharmacokinetics of oral pantoprazole in healthy volunteers.

Barbara Gawrońska-Szklarz et al.

European journal of clinical pharmacology, 68(9), 1267-1274 (2012-03-16)

Pantoprazole is metabolized by cytochrome P450 2 C19, which shows genetic polymorphism. The effect of CYP2C19 polymorphism on single-dose pharmacokinetics of oral pantoprazole in healthy volunteers was evaluated. Pantoprazole pharmacokinetics was determined in 32 healthy volunteers after a 40-mg single

Polystyrene microgel amphiphiles with maltohexaose. Synthesis, characterization, and potential applications.

Atsushi Narumi et al.

Biomacromolecules, 7(5), 1496-1501 (2006-05-09)

4-Vinylbenzyl maltohexaoside peracetate (1) was copolymerized with divinylbenzene (DVB) using 1-phenyl-1-(2',2',6',6'-tetramethyl-1'-piperidinyloxy)ethane (2) in m-xylene. The copolymerizations were performed at 138 degrees C for 20 h using the mole fraction of 1 in the total feed of 1 and DVB (F1:

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