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Showing 1-13 of 13 results for "87580" within Papers
Use of purified and modified bentonites in linear low-density polyethylene/organoclay/compatibilizer nanocomposites
Tijen S et al.
Journal of Applied Polymer Science, 124, 2430-2440 (2012)
Surface activity of water soluble amphiphilic chitin derivatives.
Desbrieres J
Polym. Bull., 39(2), 209-215 (1997)
Preparation of highly uniform Pickering emulsions by mercaptocarboxylated gold nanoparticles.
Yamanaka K
Colloids and Surfaces. A, Physicochemical and Engineering Aspects, 436, 18-25 (2013)
Electron tomography and 3D molecular simulations of platinum nanocrystals.
Florea I
Nanoscale, 4(16), 5125-5131 (2012)
Derivatization of saturated long-chain fatty acids with phenacyl bromide in non-ionic micelles
Ilana and Zamir
Journal of Chromatography A, 586, 347-350 (1991)
O-Alkylation of pyridine aldo-and ketoximes with dihalohydrins under phase-transfer conditions
Kocak A, et al.
Synthetic Communications, 37, 1155-1165 (2007)
Ionomers for tunable softening of thermoplastic polyurethane
Zander Z, et al.
Macromolecules, 49, 926-934 (2016)
Ghassan Bkaily et al.
Canadian journal of physiology and pharmacology, 93(11), 923-934 (2015-08-21)
Using the UM-X7.1 hereditary cardiomyopathic and muscular dystrophy hamsters (HCMH), we tested the effects of lifelong preventive or curative treatments during the heart failure phase with the NHE-1 inhibitor EMD 87580 (EMD) or with the angiotensin-converting enzyme inhibitor cilazapril on
Juliana Fantinelli et al.
European journal of pharmacology, 737, 125-132 (2014-05-27)
Previous results show that prolonged treatment with EMD-87580 (EMD) NHE-1 blocker attenuates and reverses postinfarction remodelling. Our aim was to evaluate the effects of the treatment of EMD compared to ischemic postconditioning (IPO) in a model of regional ischemia. Isolated
Sabzali Javadov et al.
Basic research in cardiology, 106(1), 99-109 (2010-10-05)
Studies on the role of mitochondrial fission/fusion (MFF) proteins in the heart have been initiated recently due to their biological significance in cell metabolism. We hypothesized that the expression of MFF proteins is affected by post-infarction remodeling and in vitro
Xiaohong Tracey Gan et al.
Cardiovascular research, 85(1), 79-89 (2009-08-19)
Cardiac glycosides induce cardiomyocyte hypertrophy via yet to be defined mechanisms. These hypertrophic effects are likely related to changes in intracellular signalling secondary to Na(+)-K(+) ATPase (NKA) inhibition which would produce elevations in intracellular sodium concentrations. Sodium-hydrogen exchanger isoform 1
Mohamed Mlih et al.
Molecular and cellular biochemistry, 404(1-2), 211-220 (2015-03-12)
Osteopontin (OPN), a multifunctional glycophosphoprotein, has been reported to contribute to the development and progression of cardiac remodeling and hypertrophy. Cardiac-specific OPN knockout mice were protected against hypertrophy and fibrosis mediated by Ang II. Recently, transgenic mice expressing the active
Ersilia Coccaro et al.
American journal of physiology. Heart and circulatory physiology, 297(2), H846-H858 (2009-06-23)
The mammalian Na(+)/H(+) exchanger isoform 1 (NHE1) is a ubiquitously expressed membrane protein that regulates intracellular pH in the myocardium. NHE1 is also important in mediating myocardial hypertrophy, and the blockage of NHE1 activity prevents hypertrophy and reduces ischemia-reperfusion injury
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