SAB2501686

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Showing 1-9 of 9 results for "SAB2501686" within Papers

LRRC8A channels support TNFα-induced superoxide production by Nox1 which is required for receptor endocytosis.

Hyehun Choi et al.

Free radical biology & medicine, 101, 413-423 (2016-11-14)

Leucine Rich Repeat Containing 8A (LRRC8A) is a required component of volume-regulated anion channels (VRACs). In vascular smooth muscle cells, tumor necrosis factor-α (TNFα) activates VRAC via type 1 TNFα receptors (TNFR1), and this requires superoxide (O2•-) production by NADPH

Phosphorylation of Nox1 regulates association with NoxA1 activation domain.

Jennifer Streeter et al.

Circulation research, 115(11), 911-918 (2014-09-18)

Activation of Nox1 initiates redox-dependent signaling events crucial in the pathogenesis of vascular disease. Selective targeting of Nox1 is an attractive potential therapy, but requires a better understanding of the molecular modifications controlling its activation. To determine whether posttranslational modifications

NADPH oxidases in Parkinson?s disease: a systematic review

Karim Belarbi

Molecular Neurobiology, 12(84) (2017)

Nox1 NADPH oxidase is necessary for late but not early myocardial ischaemic preconditioning

Shuxia Jiang

Cardiovascular Research, 102(1) (2014)

LRRC8A channels support TNFa-induced superoxide production by Nox1 which is required for receptor endocytosis

Hyehun Choi

Free Radical Biology & Medicine, 413-423 (2016)

TRPA1 mediates damage of the retina induced by ischemia and reperfusion in mice.

Daniel Souza Monteiro de Araújo et al.

Cell death & disease, 11(8), 633-633 (2020-08-18)

Oxidative stress is implicated in retinal cell injury associated with glaucoma and other retinal diseases. However, the mechanism by which oxidative stress leads to retinal damage is not completely understood. Transient receptor potential ankyrin 1 (TRPA1) is a redox-sensitive channel

Reactive oxygen generated by Nox1 triggers the angiogenic switch

Jack L

Proceedings of the National Academy of Sciences of the USA, 99(2), 715-720 (2000)

Nox1 NADPH oxidase is necessary for late but not early myocardial ischaemic preconditioning.

Shuxia Jiang et al.

Cardiovascular research, 102(1), 79-87 (2014-02-07)

Ischaemic preconditioning (IPC) is an adaptive mechanism that renders the myocardium resistant to injury from subsequent hypoxia. Although reactive oxygen species (ROS) contribute to both the early and late phases of IPC, their enzymatic source and associated signalling events have

Distinct Subcellular Localizations of Nox1 and Nox4 in Vascular Smooth Muscle Cells

Lula L

Arteriosclerosis, Thrombosis, and Vascular Biology, 24(4) (2004)

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