SAB4200195

Applied Filters:

Keyword:'SAB4200195'

Showing 1-10 of 10 results for "SAB4200195" within Papers

SMARCA4 (BRG1) loss of expression is a useful marker for the diagnosis of ovarian small cell carcinoma of the hypercalcemic type (ovarian rhabdoid tumor): a comprehensive analysis of 116 rare gynecologic tumors, 9 soft tissue tumors, and 9 melanomas.

Karanian-Philippe M

American Journal of Surgical Pathology, 39, 1197-1205 (2015)

Mutations affecting components of the SWI/SNF complex cause Coffin-Siris syndrome.

Tsurusaki Y

Nature Genetics, 44, 376-378 (2012)

BRG1 variant rs1122608 on chromosome 19p13.2 confers protection against stroke and regulates expression of pre-mRNA-splicing factor SFRS3

Xiong X

Human Genetics, 133, 499-508 (2014)

Oxidative stress and hepatic stellate cell activation are key events in arsenic induced liver fibrosis in mice.

Subhadip Ghatak et al.

Toxicology and applied pharmacology, 251(1), 59-69 (2010-12-08)

Arsenic is an environmental toxicant and carcinogen. Exposure to arsenic is associated with development of liver fibrosis and portal hypertension through ill defined mechanisms. We evaluated hepatic fibrogenesis after long term arsenic exposure in a murine model. BALB/c mice were

Brg1, the ATPase subunit of the SWI/SNF chromatin remodeling complex, is required for myeloid differentiation to granulocytes.

Vradii D

Journal of Cellular Physiology, 206, 112-118 (2006)

The BRG1 transcriptional coregulator

Trotter KW and Archer TK.

Nuclear Receptor Signaling (2008)

Germline nonsense mutation and somatic inactivation of SMARCA4/BRG1 in a family with rhabdoid tumor predisposition syndrome.

Schneppenheim R

American Journal of Human Genetics, 86, 279-284 (2010)

CTCF binds to sites in the major histocompatibility complex that are rapidly reconfigured in response to interferon-gamma.

Diego Ottaviani et al.

Nucleic acids research, 40(12), 5262-5270 (2012-03-01)

Activation of the major histocompatibility complex (MHC) by interferon-gamma (IFN-γ) is a fundamental step in the adaptive immune response to pathogens. Here, we show that reorganization of chromatin loop domains in the MHC is evident within the first 30 min

BRG1 co-localizes with DNA replication factors and is required for efficient replication fork progression.

Stephanie M Cohen et al.

Nucleic acids research, 38(20), 6906-6919 (2010-06-24)

For DNA replication to occur, chromatin must be remodeled. Yet, we know very little about which proteins alter nucleosome occupancy at origins and replication forks and for what aspects of replication they are required. Here, we demonstrate that the BRG1

Exit from G1 and S phase of the cell cycle is regulated by repressor complexes containing HDAC-Rb-hSWI/SNF and Rb-hSWI/SNF.

H S Zhang et al.

Cell, 101(1), 79-89 (2000-04-25)

We present evidence that Rb forms a repressor containing histone deacetylase (HDAC) and the hSWI/SNF nucleosome remodeling complex, which inhibits transcription of genes for cyclins E and A and arrests cells in the G1 phase of the cell cycle. Phosphorylation

Page 1 of 1