G4635
Gitoxigenin
Synonym(e):
3,14,16β,21-Tetrahydroxy-nor-20(22)-cholensäurelacton
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Empirische Formel (Hill-System):
C23H34O5
CAS-Nummer:
Molekulargewicht:
390.51
PubChem Substance ID:
UNSPSC Code:
12352200
EC Number:
208-886-2
MDL number:
Beilstein/REAXYS Number:
96483
Technischer Dienst
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Unterstützung erhaltenstorage temp.
−20°C
SMILES string
[H][C@]12CC[C@]3([H])[C@]([H])(CC[C@]4(C)[C@H]([C@@H](O)C[C@]34O)C5=CC(=O)OC5)[C@@]1(C)CC[C@H](O)C2
Biochem/physiol Actions
Structure-activity relationships for 16β-substituted digitoxigenins as inhibitors of Na+/K+ ATPases.
signalword
Danger
hcodes
Hazard Classifications
Acute Tox. 3 Dermal - Acute Tox. 3 Inhalation - Acute Tox. 3 Oral
Lagerklasse
6.1C - Combustible acute toxic Cat.3 / toxic compounds or compounds which causing chronic effects
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
ppe
Eyeshields, Faceshields, Gloves, type P2 (EN 143) respirator cartridges
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Tomonori Arao et al.
Journal of analytical toxicology, 26(4), 222-227 (2002-06-11)
Simultaneous determination of oleandrin and its three related compounds, desacetyloleandrin, oleandrigenin, and gitoxigenin in blood by using liquid chromatography-three-dimensional quadrupole mass spectrometry (LC-3DQMS) system equipped with sonic spray ionization (SSI) interface was conducted. This analyzing method was suitable for all
Miguel López-Lázaro et al.
Planta medica, 69(8), 701-704 (2003-10-08)
Recent research has shown the anticancer effects of digitalis compounds suggesting their possible use in medical oncology. Four extracts obtained from the leaves of Digitalis purpurea subsp. heywoodii have been assessed for cytotoxic activity against three human cancer cell lines
Jack T Rogers et al.
Journal of neural transmission (Vienna, Austria : 1996), 118(3), 493-507 (2011-01-12)
Increased brain α-synuclein (SNCA) protein expression resulting from gene duplication and triplication can cause a familial form of Parkinson's disease (PD). Dopaminergic neurons exhibit elevated iron levels that can accelerate toxic SNCA fibril formation. Examinations of human post mortem brain