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Merck

85451

β-Sitosterol

≥70%

Sinónimos:

α-Dihydrofucosterol, 22,23-Dihydrostigmasterol, 24α-Ethylcholesterol, 5-Stigmasten-3β-ol

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100 G

MXP 3,016.00

MXP 3,016.00

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Fórmula empírica (notación de Hill):
C29H50O
Número CAS:
83-46-5
Peso molecular:
414.71
UNSPSC Code:
12352211
NACRES:
NA.77
PubChem Substance ID:
329769380
EC Number:
201-480-6
Beilstein/REAXYS Number:
1916165
MDL number:
MFCD00003631

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InChI key

KZJWDPNRJALLNS-VJSFXXLFSA-N

InChI

1S/C29H50O/c1-7-21(19(2)3)9-8-20(4)25-12-13-26-24-11-10-22-18-23(30)14-16-28(22,5)27(24)15-17-29(25,26)6/h10,19-21,23-27,30H,7-9,11-18H2,1-6H3/t20-,21-,23+,24+,25-,26+,27+,28+,29-/m1/s1

SMILES string

O[C@H](C1)CC[C@@]2(C)C1=CC[C@]3([H])[C@]2([H])CC[C@@]4(C)[C@@]3([H])CC[C@]4([H])[C@H](C)CC[C@H](C(C)C)CC

biological source

plant

assay

≥70%

form

powder

impurities

campesterol and β-sitostanol, residual

mp

136-140 °C (lit.)

functional group

hydroxyl

Quality Level

200

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1 of 4

Este artículo
S12701612947PHR2693
β-Sitosterol ≥70%

Sigma-Aldrich

85451

β-Sitosterol

β-Sitosterol synthetic, ≥95%

Sigma-Aldrich

S1270

β-Sitosterol

β-Sitosterol United States Pharmacopeia (USP) Reference Standard

USP

1612947

β-Sitosterol

β-Sitosterol certified reference material, pharmaceutical secondary standard

Supelco

PHR2693

β-Sitosterol

assay

≥70%

assay

≥95%

assay

-

assay

-

biological source

plant

biological source

synthetic

biological source

-

biological source

-

functional group

hydroxyl

functional group

hydroxyl

functional group

-

functional group

-

form

powder

form

powder

form

-

form

solid

mp

136-140 °C (lit.)

mp

136-140 °C (lit.)

mp

136-140 °C (lit.)

mp

136-140 °C (lit.)

Quality Level

200

Quality Level

100

Quality Level

-

Quality Level

300

Application

β-Sitosterol was used to supplement diet of mice to study its effect on body mass accumulation.

Biochem/physiol Actions

β-Sitosterol is a plant sterol that has hypocholesterolemic activity.[1] Only 5% of β-Sitosterol is absorbed by humans. It is hypothesized that the cholesterol-lowering activity is by competing cholesterol for uptake by the cells or by interfering with the esterification of cholesterol.[2] β-Sitosterol is reported to reduce the incidence of colon tumors.[3]

Clase de almacenamiento

11 - Combustible Solids

wgk

WGK 3

flash_point_f

Not applicable

flash_point_c

Not applicable

ppe

Eyeshields, Gloves, type N95 (US)

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Certificados de análisis (COA)

Lot/Batch Number
BCCN6475
BCCL2725
BCCK6133
BCCJ3218
BCCH0450

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Pauline Seguinot et al.
International journal of food microbiology, 316, 108441-108441 (2019-11-30)
Even though non-Saccharomyces yeasts were regarded as spoilage microorganisms for a long time, their abilities to improve and diversify the aromatic profile of wines are now well recognized. Consequently, their use in combination with S. cerevisiae strains during winemaking has
M Sugano et al.
The Journal of nutrition, 107(11), 2011-2019 (1977-11-01)
The hypocholesterolemic activity of beta-sitosterol and its hydrogenated product, beta-sitostanol (dihydrositosterol or stigmastanol) has been compared in young male rats. When cholesterol was included in the diet, sitostanol consistently exhibited significantly greater hypocholesterolemic activity than sitosterol. There were no apparent
Yoshihide Yamanashi et al.
The Journal of pharmacology and experimental therapeutics, 320(2), 559-564 (2006-12-01)
Previous in vivo studies including those with knockout mice suggested that Niemann-Pick C1-like 1 (NPC1L1) plays an essential role in the intestinal absorption of cholesterol. To characterize the mechanism of cholesterol uptake mediated by NPC1L1, an in vitro system reflecting
Pauline Seguinot et al.
Frontiers in microbiology, 11, 305-305 (2020-03-19)
Non-Saccharomyces yeasts are currently widely used in winemaking to enhance aroma profile diversity among wines. The use of Metschnikowia pulcherrima in sequential inoculation with S. cerevisiae was compared to the inoculation of a pure culture of S. cerevisiae. Moreover, various
Dietary supplementation with phytosterol and ascorbic acid reduces body mass accumulation and alters food transit time in a diet-induced obesity mouse model
Thornton SJ et al
Lipids in Health and Disease, 10, doi: 10-doi: 10 (2011)
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