SCP0134
Endothelin Antagonist PD 142893
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1 MG
190,00 €
5 MG
694,00 €
190,00 €
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Informazioni su questo articolo
Formula empirica (notazione di Hill):
C50H65N7O10
Peso molecolare:
924.09
NACRES:
NA.32
UNSPSC Code:
12352200
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Servizio Tecnico
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Permettici di aiutartiassay
≥95% (HPLC)
form
lyophilized
composition
Peptide Content, ≥85%
storage condition
protect from light
storage temp.
−20°C
1 of 4
Questo articolo | SCP0137 | SCP0143 | SCP0136 |
|---|---|---|---|
| assay ≥95% (HPLC) | assay ≥95% (HPLC) | assay ≥95% (HPLC) | assay ≥95% (HPLC) |
| form lyophilized | form lyophilized | form lyophilized | form lyophilized |
| storage condition protect from light | storage condition protect from light | storage condition protect from light | storage condition protect from light |
| storage temp. −20°C | storage temp. −20°C | storage temp. −20°C | storage temp. −20°C |
| composition Peptide Content, ≥85% | composition Peptide Content, ≥85% | composition Peptide Content, ≥75% | composition Peptide Content, ≥67% |
Application
PD 142-893 is used as a nonselective endothelin ET(A)/ET(B) receptor antagonist.
Classe di stoccaggio
11 - Combustible Solids
wgk
WGK 3
flash_point_f
Not applicable
flash_point_c
Not applicable
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Chumpon Wilasrusmee et al.
Surgery, 134(2), 384-389 (2003-08-30)
We have previously shown that endothelial injury by cyclosporin A (CyA) is associated with an increased endothelin-1 (ET-1) release. We now sought to determine, in an animal model of angiogenesis, if inhibiting the effect of ET-1 on endothelial cells (ECs)
Ashley M Stokes et al.
Canadian journal of veterinary research = Revue canadienne de recherche veterinaire, 70(3), 197-205 (2006-07-21)
The goals of this study were to determine the concentration-response (C-R) relationship of endothelin-1 (ET-1), compare 2 ET-receptor antagonists and determine the antagonist concentrations that block the vasomotor effects of ET-1, and compare the effectiveness of ET-1 and previously studied
Sebastien Faure et al.
Journal of vascular research, 43(1), 19-26 (2005-10-29)
The effect of angiotensin IV (AngIV) was studied in freshly isolated rat basilar arteries (BAs) perfused at a constant rate. AngIV had no effect on basal BA perfusion pressure, but induced a marked concentration-dependent contraction in vessels precontracted by a
Alexandra Madsen et al.
Journal of molecular and cellular cardiology, 154, 115-123 (2021-02-15)
The role of DNA methylation in cardiomyocyte physiology and cardiac disease remains a matter of controversy. We have recently provided evidence for an important role of DNMT3A in human cardiomyocyte cell homeostasis and metabolism, using engineered heart tissue (EHT) generated
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