MAB344

Anti-O1 Antibody, clone 59

Name: Anti-O1 Antibody, clone 59
Brand: MM
Price: 592 USD

clone 59, Chemicon^®, from mouse

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50 μG

$592.00

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About This Item

UNSPSC Code:

12352203

eCl@ss:

32160702

NACRES:

NA.41

Key Documents

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biological source

mouse

Quality Level

100

antibody form

saturated ammonium sulfate (SAS) precipitated

clone

59, monoclonal

species reactivity

chicken, rat, mouse, human

manufacturer/tradename

Chemicon^®

technique(s)

immunohistochemistry: suitable

isotype

IgM

shipped in

wet ice

target post-translational modification

unmodified

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Show Differences

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This Item	AB3768	AB5714	MAB4381
Sigma-Aldrich MAB344 Anti-O1 Antibody, clone 59	Sigma-Aldrich AB3768 Anti-Rad54L Antibody	Sigma-Aldrich AB5714 Anti-Hey 1/HRT1 Antibody	Sigma-Aldrich MAB4381 Anti-TRA-1-81 Antibody, clone TRA-1-81
antibody form saturated ammonium sulfate (SAS) precipitated	antibody form saturated ammonium sulfate (SAS) precipitated	antibody form saturated ammonium sulfate (SAS) precipitated	antibody form saturated ammonium sulfate (SAS) precipitated
clone 59, monoclonal	clone polyclonal	clone polyclonal	clone TRA-1-81, monoclonal
species reactivity chicken, rat, mouse, human	species reactivity human, mouse	species reactivity human, mouse	species reactivity human
biological source mouse	biological source rabbit	biological source rabbit	biological source mouse
shipped in wet ice	shipped in dry ice	shipped in dry ice	shipped in wet ice
Quality Level 100	Quality Level 100	Quality Level 100	Quality Level 100

Immunogen

White matter of corpus callosum from rat brain.

Application

Anti-O1 Antibody, clone 59 is an antibody against O1 for use in IH.

Immunohistochemistry: 10-20 μg/mL on unfixed, shock frozen tissue.

Note: 01 is a lipid which is released from the membrane by alcohol.

Optimal working dilutions must be determined by the end user.

Research Category
Neuroscience

Research Sub Category
Neuronal & Glial Markers

Biochem/physiol Actions

Recognizes Oligodendrocyte marker O1. Also reacts with certain galactolipids in sperm (see Additional Information library for list).

Physical form

Format: Purified

Liquid. Buffer = 0.05 M Potassium Phosphate, 0.3 M NaCl, pH 8.0 with 0.05% sodium azide.

Preparation Note

Maintain lyophilized material at +2–8°C for up to 12 months. After reconstitution maintain frozen at -20°C in undiluted aliquots for up to 6 months. Avoid repeated freeze/thaw cycles.

Analysis Note

Control
Oligodendrocyte culture, Brain

Other Notes

Concentration: Please refer to the Certificate of Analysis for the lot-specific concentration.

Legal Information

CHEMICON is a registered trademark of Merck KGaA, Darmstadt, Germany

Disclaimer

Unless otherwise stated in our catalog or other company documentation accompanying the product(s), our products are intended for research use only and are not to be used for any other purpose, which includes but is not limited to, unauthorized commercial uses, in vitro diagnostic uses, ex vivo or in vivo therapeutic uses or any type of consumption or application to humans or animals.

Storage Class

10 - Combustible liquids

wgk_germany

WGK 2

flash_point_f

Not applicable

flash_point_c

Not applicable

Certificates of Analysis (COA)

Search for Certificates of Analysis (COA) by entering the products Lot/Batch Number. Lot and Batch Numbers can be found on a product’s label following the words ‘Lot’ or ‘Batch’.

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[Disturbed maturation of oligodendrocyte progenitors in lipopolysaccharide-induced hypomyelination in cultured forebrain slices of neonatal rats].

Jong-Wan Kim et al.

Folia neuropathologica, 57(1), 24-35 (2019-05-01)

This study was performed to determine whether the disturbed maturation of oligodendrocyte (OL) progenitors might be related to lipopolysaccharide (LPS)-induced hypomyelination. We created organotypic cultures of forebrain slices from neonatal rats and explored the morphological changes of glial cells expressing

Olfactory ensheathing cells exhibit unique migratory, phagocytic, and myelinating properties in the X-irradiated spinal cord not shared by Schwann cells.

Karen L Lankford,Masanori Sasaki,Christine Radtke,Jeffery D Kocsis

Glia null

Progesterone Changes VEGF and BDNF Expression and Promotes Neurogenesis After Ischemic Stroke.

Chao Jiang et al.

Molecular neurobiology (2016-01-10)

Studies have shown that progesterone enhances functional recovery after ischemic stroke, but the underlying mechanisms are not completely understood. Therefore, we investigated the effect of progesterone on vascular endothelial growth factor (VEGF), brain-derived neurotrophic factor (BDNF), and neurogenesis in a

Immuno-electron-microscopic identification of O-antigen-bearing oligodendroglial cells in vitro

Berg, G and Schachner, M

Cell and Tissue Research, 219, 313-325 (1981)

Generation of functional neurons and glia from multipotent adult mouse germ-line stem cells.

Streckfuss-Bomeke, Katrin, et al.

Stem Cell Research, 2, 139-154 (2009)

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